The significant loss of blood calcium commonly characterizes the condition of hypocalcemia. In particular, the loss of calcium is common in the extracellular fluid, and it occurs in greater quantities though it is replaceable by the intestine or the bone (Watad et al., 2016). The condition is a consequence of various clinical entities with the signs and symptoms of neuromuscular irritability. The common conditions of the neuromuscular irritability include the paresthesia, laryngospasm, muscle cramps, seizures as well as tetany.
The same instability of the neuromuscular structures appears in the elicitation of the Chvostek’s signs and the common Trousseau’s signs famous as sidebars (Watad et al., 2016). The signs are the famous time honors predictions that are organized and well-chronicled the history of the medical condition with a close association with hypocalcemia.
Hypocalcemia tends to lead to the twitching of the ipsilateral facial muscles that lead to the suggestion of the neuromuscular excitability commonly associated with hypocalcemia (Watad et al., 2016). Therefore, the expected findings of the nurse assessment of the patient will lead to the two common signs of the disorder, which are the Chvostek’ sign and the Trousseau’s sign. Either of the two signs is common with the patients in the condition of hypocalcemia.
One of the common signs of Chvostek includes twitching of the facial muscles. However, the most common sign of the Trousseau’s symptom is the presence of the carpopedal spasm that takes place after a few minutes involving the inflation of sphygmomanometer cuff that occurs above the systolic blood pressure (Watad et al., 2016).
Hypocalcemia is a disorder known to cause the excitability of the nerves especially at the arm and the forearm. The process will lead to the contraction of muscles, which further goes ahead through an exacerbation process induced by the ischemia process caused by the sphygmomanometer (Batra & Agarwal, 2016). Eventually, the entire chronically effected process will lead to the twitching, an act that defines the sign in detail.
On the other hand, the administration of Vitamin D3 is vital in the process; because research shows that the vitamin is administered to, the patient can lead to the reduction of the symptomatic postoperative hypocalcemia (Batra & Agarwal, 2016). The vitamin D supplementation is also effective in preventing the symptomatic post-thyroidectomy hypocalcemia. In the current context, research suggests that the oral administration of calcium is effective for the patients under the condition of thyroidectomy and the administration of vitamin D is recommended for high-risk individuals.
In regards to the education of the patient, the RN will mainly focus on the two key points that are vital for the recovery process for the patient. Firstly, the nurse will take the patient through the procedures for adding calcium into their diet, to enable them to go about the process at home (Batra & Agarwal, 2016). Most of the time the patient with hypocalcemia are returned home after medication, and they are given a package of the drugs and the supplements to enable them to go about the process of administering the medication on their own.
Finally, the RN will teach the patient the specific diets that contain calcium, to enable them to consume such diets in reasonable quantities for a better improvement of the levels of calcium in their blood (Batra & Agarwal, 2016). Most patients have no enough information, and especially the issues about medication, are left to the advice of a health specialist, due to the sensitivity of the health status of an individual.
Batra, C. M., & Agarwal, R. (2016). Hypocalcemic Cardiomyopathy and Pseudohypoparathyroidism Due to Severe Vitamin D Deficiency. Journal of The Association of Physicians of India, 64, 74.
Watad, A., Tiosano, S., Azrielant, S., Whitby, A., Comaneshter, D., Cohen, A. D., & Amital, H. (2016). Low levels of calcium or vitamin D, which is more important in systemic lupus erythematosus patients? An extensive data analysis. Clinical and experimental rheumatology.